Quantum Computing for Disease Treatments

Quantum Computing for Disease TreatmentsQuantum Computing for Disease TreatmentsQuantum Computing for Disease Treatments

Quantum Computing for Disease Treatments

Quantum Computing for Disease TreatmentsQuantum Computing for Disease TreatmentsQuantum Computing for Disease Treatments

Our goal is to find the best treatment for patients using quantum computing

Our goal is to find the best treatment for patients using quantum computingOur goal is to find the best treatment for patients using quantum computingOur goal is to find the best treatment for patients using quantum computing

Our goal is to find the best treatment for patients using quantum computing

Our goal is to find the best treatment for patients using quantum computingOur goal is to find the best treatment for patients using quantum computingOur goal is to find the best treatment for patients using quantum computing

OUR WORK (COMPUTATIONAL ,SCORING PERFORMED W/ AUTODOCKVINA)

TOP BINDING TARGETS AND SCORES...SOME EVEN BIND TO MULTIPLE TARGETS!!

AS OF March 27 2026, WE HAVE 96 ELITE MOLECULES BINDING -10 OR STRONGER AND 480 STRONG BINDERS AT -9 AND STRONGER!! ( some targets i have hit with multiple molecules binding strenght -9.99 and stronger!!!

FAK (focial adhesion kinease

-11.04. My new strongest fak molecule!

4K9Y (FAK Kinase): Focal adhesion kinase. Overactive in cancer, promoting survival and metastasis. Drugs must inhibit the ATP-binding pocket to block its signaling. This structure shows a fragment inhibitor bound there .

DNA GYRASE

-9.86

· Bacterial (DNA Gyrase): Improper function = essential bacterial enzyme. Drug need = inhibitors that are selective for bacterial vs human enzymes.

EGFR KINASE

-9.84

 Improper function = overactivity. Drug need = ATP-competitive inhibitors.

Human renin-9.59

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BCL-2 -9.24

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6LU7 (SARS-CoV-2 Mpro) -7.59

Main protease. Virus needs this enzyme to replicate. Drugs must block the catalytic site (His41/Cys145) to stop replication . The drug that i created that has the top score summary, Structure A fluorinated polycyclic aromatic with a carboxylic acid and hydroxyl group and a MW: 232.21. it has a shallow, solvent-exposed active site that's harder for small molecules to grip onto compared to deeper pockets like ACE or FAK. That's actually why Paxlovid (Nirmatrelvir) needs a covalent warhead to bind

ACE

AS OF 3/27/26 I HAVE FOUND MY STRONGEST BINDER SO FAR AND FOR ACE..... -13.27. AN UNBELIEVEABLE #!!

Angiotensin-converting enzyme. Produces angiotensin II, raising blood pressure. Inhibitors (Lisinopril, Enalapril) block the active site to lower BP .  As of Feb 23 2026 we have a new high , the new ace molecule has a score of -10.95, is my new strongest binding molecule to date, and puts me .05 closer to -11 territory!!!!

Dopamine D2

-9.83

Improper function = over/under signaling. Drug need = antagonists or agonists.

SARS COV-2 SPIKE -10.22!!!!!

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Contact Us

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3323233477 and Quantumcomputingdiseasecures@gmail.com

Breaking Boundaries: Some of My Work in Pictures Also my top binding scores for all targets currently and scores get better constantly !!!!!!!

    SOME OF OUR CURRENT TARGETS( OF ABOUT 25)

    CDK2 ( CYLIN-DEPENDENT KINEASE 2 -7.68 ( 3 19 26) up to -9.35 new discovery!!!!!

    THIS IS A CRITICAL CELL CYCLE REGULATOR.

    BECAUSE IT IS HYPERACTIVATED IN MANY CANCERS, IT IS A MAJOR TARGET FOR THERAPEUTIC INHIBITORS. 

    (BRD4) -9.52

    Improper function = abnormal gene expression. Drug need = acetyl-lysine competitive inhibitors

    DNA GYRASE -9.86

    WHY ITS VITAL: WITHOUT GYRASE, THE DNA WOULD BEXOME SO TIGHTLY KNOTTED( POSITIVE SUPERCOILING) THAT THE CELL COULD NOT REPLICATE ITS GENOME OR TRANSCRIBE GENES, LEADING TO CELL DEATH!

    SARS-CoV-2 Mpro -7.59

    Main protease. Virus needs this enzyme to replicate. Drugs must block the catalytic site (His41/Cys145) to stop replication .

    DPP-4, Insulin Receptor -9.38( 3 19 26) new small molecule binding insulin receptor -9.49!!!!!!

    Improper function = hormone dysregulation. Drug need = enzyme inhibitors (DPP-4) or receptor modulators (Insulin).

    GABA-A/GlyR -7.08

    Improper function = neuronal hyperexcitability. Drug need = positive allosteric modulators to enhance inhibitory signals.

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